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The Effect of NO and CO on Cellular Iron Metabolism: A comparison of two diatomic cell signalling molecules - Softcover
Nitric oxide (NO) and carbon monoxide (CO) are toxic gases in the atmosphere that are usually associated with pollution. Surprisingly, both have been found to be synthesised within the body and have functions as signalling molecules. It has been shown that they actuate their signal by interacting with iron (Fe) in Fe-containing proteins. Fe is an obligate requirement for cells as many important enzymatic proteins utilise this element for redox activity. Activated macrophages produce large quantities of NO and it has been hypothesised that one of the cytotoxic mechanisms of activated macrophages is the NO crossing into the target cell and removing the Fe from the proteins and effluxing it from the cell. CO also interacts with Fe, but its effects on cellular Fe metabolism have not been examined in as great detail as NO has. This work has examined the effects of NO donors and CO gas and donors on cellular Fe metabolism. It describes the pathway NO utilises in its removal of Fe from cells and compares these results with CO. It indicates that although both molecules interact with Fe they have different effects on cellular Fe metabolism.
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About the Author:
Dr Watts graduated with a PhD from the University of New South Wales in 2004. He currently holds a Peter Doherty Research Fellowship from the National Health and Medical Research Council Australia (NHMRC). Prof Richardson is Professor of Cancer Cell Biology and NHMRC Senior Principal Research Fellow at the University of Sydney, Australia.
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- PublisherVDM Verlag
- Publication date2009
- ISBN 10 3639146581
- ISBN 13 9783639146585
- BindingPaperback
- Number of pages196
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The Effect of NO and CO on Cellular Iron Metabolism: A comparison of two diatomic cell signalling molecules
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Book Description Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Nitric oxide (NO) and carbon monoxide (CO) are toxic gases in the atmosphere that are usually associated with pollution. Surprisingly, both have been found to be synthesised within the body and have functions as signalling molecules. It has been shown that they actuate their signal by interacting with iron (Fe) in Fe-containing proteins. Fe is an obligate requirement for cells as many important enzymatic proteins utilise this element for redox activity. Activated macrophages produce large quantities of NO and it has been hypothesised that one of the cytotoxic mechanisms of activated macrophages is the NO crossing into the target cell and removing the Fe from the proteins and effluxing it from the cell. CO also interacts with Fe, but its effects on cellular Fe metabolism have not been examined in as great detail as NO has. This work has examined the effects of NO donors and CO gas and donors on cellular Fe metabolism. It describes the pathway NO utilises in its removal of Fe from cells and compares these results with CO. It indicates that although both molecules interact with Fe they have different effects on cellular Fe metabolism. 196 pp. Englisch. Seller Inventory # 9783639146585
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The Effect of NO and CO on Cellular Iron Metabolism
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Book Description Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Watts RalphDr Watts graduated with a PhD from the University of New South nWales in 2004. He currently holds a Peter Doherty Research nFellowship from the National Health and Medical Research Council nAustralia (NHMRC). Prof Richards. Seller Inventory # 151365638
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The Effect of NO and CO on Cellular Iron Metabolism : A comparison of two diatomic cell signalling molecules
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ISBN 13: 9783639146585
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Book Description Taschenbuch. Condition: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Nitric oxide (NO) and carbon monoxide (CO) are toxic gases in the atmosphere that are usually associated with pollution. Surprisingly, both have been found to be synthesised within the body and have functions as signalling molecules. It has been shown that they actuate their signal by interacting with iron (Fe) in Fe-containing proteins. Fe is an obligate requirement for cells as many important enzymatic proteins utilise this element for redox activity. Activated macrophages produce large quantities of NO and it has been hypothesised that one of the cytotoxic mechanisms of activated macrophages is the NO crossing into the target cell and removing the Fe from the proteins and effluxing it from the cell. CO also interacts with Fe, but its effects on cellular Fe metabolism have not been examined in as great detail as NO has. This work has examined the effects of NO donors and CO gas and donors on cellular Fe metabolism. It describes the pathway NO utilises in its removal of Fe from cells and compares these results with CO. It indicates that although both molecules interact with Fe they have different effects on cellular Fe metabolism. Seller Inventory # 9783639146585
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