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Tackling a Legendary Scientific Mystery

Ph. D. , Mba, Roderick Slavcev

ISBN 10: 383831963X / ISBN 13: 9783838319636
Published by LAP Lambert Academic Publishing
New Condition: New Soft cover
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Paperback. 264 pages. Dimensions: 8.7in. x 5.9in. x 0.6in.The T4rII exclusion (Rex) Phenotype is defined as the inability of rII mutants of bacteriophage T4 to generate plaques on a cell lawn of E. coli harboring a stably integrated lambda prophage. The selective exclusion of the rII mutants of T4 played a critical role in our understanding of modern molecular genetics. Shockingly, since first exploited by Seymour Benzer in the mid 1950s, the mechanism underpinning the Rex phenotype remains unexplained. T4rII exclusion is encoded by the lambda genes rexA-rexB co-expressed with the lambda repressor from the prophage as the Pm-cI-rexA-rexB-timm operon. The onset of exclusion can result in the shutdown of cellular metabolism, preventing the replication of the super-infecting T4rII bacteriophage. In addition to reviewing the historic plight to characterize and resolve the Rex phenotype, we tackle this enigmatic phenomenon, adding important pieces to this puzzle and offer a working model for the mechanism of action. This item ships from multiple locations. Your book may arrive from Roseburg,OR, La Vergne,TN. Bookseller Inventory # 9783838319636

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Title: Tackling a Legendary Scientific Mystery

Publisher: LAP Lambert Academic Publishing

Binding: Paperback

Book Condition:New

Book Type: Paperback

About this title

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The T4rII exclusion (Rex) Phenotype is defined as the inability of rII mutants of bacteriophage T4 to generate plaques on a cell lawn of E. coli harboring a stably integrated lambda prophage. The selective exclusion of the rII mutants of T4 played a critical role in our understanding of modern molecular genetics. Shockingly, since first exploited by Seymour Benzer in the mid 1950's, the mechanism underpinning the Rex phenotype remains unexplained. T4rII exclusion is encoded by the lambda genes rexA-rexB co-expressed with the lambda repressor from the prophage as the Pm-cI-rexA-rexB-timm operon. The onset of exclusion can result in the shutdown of cellular metabolism, preventing the replication of the super-infecting T4rII bacteriophage. In addition to reviewing the historic plight to characterize and resolve the Rex phenotype, we tackle this enigmatic phenomenon, adding important pieces to this puzzle and offer a working model for the mechanism of action.

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