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McGraw-Hill

Published by McGraw-Hill

ISBN 10: 0390299499 ISBN 13: 9780390299499

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About this Item: McGraw-Hill. Paperback. Condition: VERY GOOD. Light rubbing wear to cover, spine and page edges. Very minimal writing or notations in margins not affecting the text. Possible clean ex-library copy, with their stickers and or stamp(s). Seller Inventory # 2808065755

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McGraw-Hill

Published by McGraw-Hill

ISBN 10: 0390299499 ISBN 13: 9780390299499

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About this Item: McGraw-Hill. Paperback. Condition: VERY GOOD. Light rubbing wear to cover, spine and page edges. Very minimal writing or notations in margins not affecting the text. Possible clean ex-library copy, with their stickers and or stamp(s). Seller Inventory # 2894946655

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McGraw-Hill

Published by McGraw-Hill

ISBN 10: 0390299499 ISBN 13: 9780390299499

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About this Item: McGraw-Hill. Paperback. Condition: VERY GOOD. Light rubbing wear to cover, spine and page edges. Very minimal writing or notations in margins not affecting the text. Possible clean ex-library copy, with their stickers and or stamp(s). Seller Inventory # 2927384654

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McGraw-Hill

Published by McGraw-Hill

ISBN 10: 0390299499 ISBN 13: 9780390299499

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About this Item: McGraw-Hill. Paperback. Condition: VERY GOOD. Light rubbing wear to cover, spine and page edges. Very minimal writing or notations in margins not affecting the text. Possible clean ex-library copy, with their stickers and or stamp(s). Seller Inventory # 2927384655

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McGraw-Hill

Published by McGraw-Hill (2010)

ISBN 10: 0390299499 ISBN 13: 9780390299499

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About this Item: McGraw-Hill, 2010. Paperback. Condition: Good. Satisfaction 100% guaranteed. Seller Inventory # mon0000786054

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McGraw-Hill

Published by McGraw-Hill

ISBN 10: 0390299499 ISBN 13: 9780390299499

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About this Item: McGraw-Hill. Paperback. Condition: Good. A copy that has been read, but remains in clean condition. All pages are intact, and the cover is intact. The spine may show signs of wear. Pages can include limited notes and highlighting, and the copy can include previous owner inscriptions. At ThriftBooks, our motto is: Read More, Spend Less. Seller Inventory # G0390299499I3N00

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Overveld, Petra Van

Published by Graphic Support, Dreumel (2005)

ISBN 10: 9090193707 ISBN 13: 9789090193700

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About this Item: Graphic Support, Dreumel, 2005. Pb. Condition: VG-. 257 pages, wrapper extremities lightly rubbed, both corners of front cover are bumped and slightly curled, affecting the pages at the beginnning of the text, top corner of front cover is creased. Seller Inventory # 134939

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McGraw Hill

Published by McGraw Hill (2010)

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About this Item: McGraw Hill, 2010. Paperback. Condition: Good. Satisfaction 100% guaranteed. Seller Inventory # mon0000763884

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Morten G. Ender, Betsy Lucal, Ralph B. McNeal, Jr., Kathleen A. Tiemann

Published by Pearson Custom Library (2014)

ISBN 10: 1269301489 ISBN 13: 9781269301480

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About this Item: Pearson Custom Library, 2014. Paperback. Condition: Acceptable. Some wear Satisfaction 100% guaranteed. Seller Inventory # mon0001658677

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Curtis W. Higgins

Published by CreateSpace Independent Publishing Platform (2013)

ISBN 10: 1482093081 ISBN 13: 9781482093087

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About this Item: CreateSpace Independent Publishing Platform, 2013. Paperback. Condition: Brand New. 1st edition. 190 pages. 9.00x6.00x0.43 inches. This item is printed on demand. Seller Inventory # zk1482093081

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Curtis W. Higgins

Published by CreateSpace Independent Publishing Platform (2013)

ISBN 10: 1482093081 ISBN 13: 9781482093087

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About this Item: CreateSpace Independent Publishing Platform, 2013. Condition: New. book. Seller Inventory # M1482093081

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Cabianca, Daphne S.

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: Condition: New. Publisher/Verlag: LAP Lambert Academic Publishing | A Polycomb/Trithorax Epigenetic Switch in Muscular Dystrophy | FSHD is an autosomal dominant disease associated with contraction of D4Z4 repeats, mapping to 4q35. By an unknown mechanism, D4Z4 deletion causes an epigenetic switch leading to de-repression of 4q35 genes. Repression by D4Z4 displays similarities with Polycomb-mediated silencing. Polycomb (PcG) and Trithorax (TrxG) group proteins work as counteractors in the epigenetic regulation of gene expression. Transcription of PcG-binding regions can regulate PcG activity. Intriguingly, it is shown here that D4Z4 generates a chromatin-bound RNA selectively in FSHD patients and upon 4q35 de-repression. This transcript is specifically 4q35-associated and is required for 4q35 gene de-repression. Interestingly, the identified RNA promotes the recruitment of the TrxG protein Ash1L to the FSHD locus. Notably, Ash1L is necessary for 4q35 gene de-repression. These results suggest that the RNA functions by recruiting Ash1L to counteract PcG silencing at D4Z4 leading to 4q35 gene de-repression in FSHD. This work provides a nexus for revealing the epigenetic basis of FSHD etiology. Moreover, this study gives insights into the biological function of repetitive elements. | Format: Paperback | Language/Sprache: english | 116 pp. Seller Inventory # K9783846528129

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Daphne S Cabianca (author)

Published by LAP Lambert Academic Publishing 2012-08-16 (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing 2012-08-16, 2012. paperback. Condition: New. Seller Inventory # 9783846528129

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Cabianca, Daphne S.

Published by LAP Lambert Academic Publishing (2016)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing, 2016. Paperback. Condition: New. PRINT ON DEMAND Book; New; Publication Year 2016; Not Signed; Fast Shipping from the UK. No. book. Seller Inventory # ria9783846528129_lsuk

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Daphne S Cabianca

Published by LAP Lambert Academic Publishing (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing, 2012. PAP. Condition: New. New Book. Delivered from our UK warehouse in 4 to 14 business days. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000. Seller Inventory # LQ-9783846528129

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Daphne S Cabianca

Published by LAP Lambert Academic Publishing (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing, 2012. PAP. Condition: New. New Book. Shipped from US within 10 to 14 business days. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000. Seller Inventory # IQ-9783846528129

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Daphne S. Cabianca

Published by LAP Lambert Academic Publishing Aug 2012 (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing Aug 2012, 2012. Taschenbuch. Condition: Neu. Neuware - FSHD is an autosomal dominant disease associated with contraction of D4Z4 repeats, mapping to 4q35. By an unknown mechanism, D4Z4 deletion causes an epigenetic switch leading to de-repression of 4q35 genes. Repression by D4Z4 displays similarities with Polycomb-mediated silencing. Polycomb (PcG) and Trithorax (TrxG) group proteins work as counteractors in the epigenetic regulation of gene expression. Transcription of PcG-binding regions can regulate PcG activity. Intriguingly, it is shown here that D4Z4 generates a chromatin-bound RNA selectively in FSHD patients and upon 4q35 de-repression. This transcript is specifically 4q35-associated and is required for 4q35 gene de-repression. Interestingly, the identified RNA promotes the recruitment of the TrxG protein Ash1L to the FSHD locus. Notably, Ash1L is necessary for 4q35 gene de-repression. These results suggest that the RNA functions by recruiting Ash1L to counteract PcG silencing at D4Z4 leading to 4q35 gene de-repression in FSHD. This work provides a nexus for revealing the epigenetic basis of FSHD etiology. Moreover, this study gives insights into the biological function of repetitive elements. 116 pp. Englisch. Seller Inventory # 9783846528129

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Daphne S. Cabianca

Published by LAP Lambert Academic Publishing Aug 2012 (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing Aug 2012, 2012. Taschenbuch. Condition: Neu. Neuware - FSHD is an autosomal dominant disease associated with contraction of D4Z4 repeats, mapping to 4q35. By an unknown mechanism, D4Z4 deletion causes an epigenetic switch leading to de-repression of 4q35 genes. Repression by D4Z4 displays similarities with Polycomb-mediated silencing. Polycomb (PcG) and Trithorax (TrxG) group proteins work as counteractors in the epigenetic regulation of gene expression. Transcription of PcG-binding regions can regulate PcG activity. Intriguingly, it is shown here that D4Z4 generates a chromatin-bound RNA selectively in FSHD patients and upon 4q35 de-repression. This transcript is specifically 4q35-associated and is required for 4q35 gene de-repression. Interestingly, the identified RNA promotes the recruitment of the TrxG protein Ash1L to the FSHD locus. Notably, Ash1L is necessary for 4q35 gene de-repression. These results suggest that the RNA functions by recruiting Ash1L to counteract PcG silencing at D4Z4 leading to 4q35 gene de-repression in FSHD. This work provides a nexus for revealing the epigenetic basis of FSHD etiology. Moreover, this study gives insights into the biological function of repetitive elements. 116 pp. Englisch. Seller Inventory # 9783846528129

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Daphne S. Cabianca

Published by LAP LAMBERT Academic Publishing (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP LAMBERT Academic Publishing, 2012. Paperback. Condition: Used: Good. Seller Inventory # SONG3846528129

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Daphne S. Cabianca

Published by LAP Lambert Academic Publishing

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing. Paperback. Condition: New. 116 pages. Dimensions: 8.7in. x 5.9in. x 0.3in.FSHD is an autosomal dominant disease associated with contraction of D4Z4 repeats, mapping to 4q35. By an unknown mechanism, D4Z4 deletion causes an epigenetic switch leading to de-repression of 4q35 genes. Repression by D4Z4 displays similarities with Polycomb-mediated silencing. Polycomb (PcG) and Trithorax (TrxG) group proteins work as counteractors in the epigenetic regulation of gene expression. Transcription of PcG-binding regions can regulate PcG activity. Intriguingly, it is shown here that D4Z4 generates a chromatin-bound RNA selectively in FSHD patients and upon 4q35 de-repression. This transcript is specifically 4q35-associated and is required for 4q35 gene de-repression. Interestingly, the identified RNA promotes the recruitment of the TrxG protein Ash1L to the FSHD locus. Notably, Ash1L is necessary for 4q35 gene de-repression. These results suggest that the RNA functions by recruiting Ash1L to counteract PcG silencing at D4Z4 leading to 4q35 gene de-repression in FSHD. This work provides a nexus for revealing the epigenetic basis of FSHD etiology. Moreover, this study gives insights into the biological function of repetitive elements. This item ships from multiple locations. Your book may arrive from Roseburg,OR, La Vergne,TN. Paperback. Seller Inventory # 9783846528129

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Daphne S Cabianca

Published by LAP Lambert Academic Publishing, Germany (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing, Germany, 2012. Paperback. Condition: New. Aufl.. Language: English. Brand new Book. FSHD is an autosomal dominant disease associated with contraction of D4Z4 repeats, mapping to 4q35. By an unknown mechanism, D4Z4 deletion causes an epigenetic switch leading to de-repression of 4q35 genes. Repression by D4Z4 displays similarities with Polycomb-mediated silencing. Polycomb (PcG) and Trithorax (TrxG) group proteins work as counteractors in the epigenetic regulation of gene expression. Transcription of PcG-binding regions can regulate PcG activity. Intriguingly, it is shown here that D4Z4 generates a chromatin-bound RNA selectively in FSHD patients and upon 4q35 de-repression. This transcript is specifically 4q35-associated and is required for 4q35 gene de-repression. Interestingly, the identified RNA promotes the recruitment of the TrxG protein Ash1L to the FSHD locus. Notably, Ash1L is necessary for 4q35 gene de-repression. These results suggest that the RNA functions by recruiting Ash1L to counteract PcG silencing at D4Z4 leading to 4q35 gene de-repression in FSHD. This work provides a nexus for revealing the epigenetic basis of FSHD etiology. Moreover, this study gives insights into the biological function of repetitive elements. Seller Inventory # AAV9783846528129

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Daphne S. Cabianca

Published by LAP LAMBERT Academic Publishing (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP LAMBERT Academic Publishing, 2012. Condition: New. book. Seller Inventory # M3846528129

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Juliana Prügel

Published by AV Akademikerverlag (2015)

ISBN 10: 3639477774 ISBN 13: 9783639477771

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About this Item: AV Akademikerverlag, 2015. Paperback. Condition: New. Language: German . Brand New Book. Die Fazioskapulohumerale Muskeldystrophie (FSHD) ist die dritthäufigste hereditäre Myopathie aus dem Bereich der neuromuskulären Erkrankungen. Sie zeichnet sich durch einen sehr unterschiedlichen Krankheitsbeginn und Krankheitsverlauf aus, wobei vor allem die Gesichts-,Schultergürtel- und Oberarmmuskulatur betroffen sind. Eine kausale Behandlung ist nicht möglich, FSHD nicht heilbar. Medikamentöse Therapien zeigen bisher keinen bemerkenswerten Erfolg und gentherapeutische Ansätze scheitern aufgrund der komplexen Pathogenese. Daher steht die symptomatische Behandlung mittels physikalisch-medizinischen Maßnahmen im Vordergrund. Aufgrund des variablen Schweregrades und großen Spektrums an Symptomen der FSHD ist eine individualisierte Physiotherapie besonders erforderlich und wichtig. Eine differenzierte Befundaufnahme und auf die Bedürfnisse des Patienten abgestimmte Behandlung spielen hierbei eine große Rolle. Wie solch ein Befund und eine entsprechende Therapie aussehen können, werden in dieser Arbeit einerseits anhand von drei Patientenbeispielen vorgestellt. Andererseits kommt eine erfahrende Physiotherapeutin zu Wort. So wird die Notwendigkeit und Bedeutung der individualisierten Physiotherapie bei FSHD eindrucksvoll verdeutlicht. Seller Inventory # KNV9783639477771

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Daphne S. Cabianca

Published by LAP Lambert Academic Publishing Aug 2012 (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP Lambert Academic Publishing Aug 2012, 2012. Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Neuware - FSHD is an autosomal dominant disease associated with contraction of D4Z4 repeats, mapping to 4q35. By an unknown mechanism, D4Z4 deletion causes an epigenetic switch leading to de-repression of 4q35 genes. Repression by D4Z4 displays similarities with Polycomb-mediated silencing. Polycomb (PcG) and Trithorax (TrxG) group proteins work as counteractors in the epigenetic regulation of gene expression. Transcription of PcG-binding regions can regulate PcG activity. Intriguingly, it is shown here that D4Z4 generates a chromatin-bound RNA selectively in FSHD patients and upon 4q35 de-repression. This transcript is specifically 4q35-associated and is required for 4q35 gene de-repression. Interestingly, the identified RNA promotes the recruitment of the TrxG protein Ash1L to the FSHD locus. Notably, Ash1L is necessary for 4q35 gene de-repression. These results suggest that the RNA functions by recruiting Ash1L to counteract PcG silencing at D4Z4 leading to 4q35 gene de-repression in FSHD. This work provides a nexus for revealing the epigenetic basis of FSHD etiology. Moreover, this study gives insights into the biological function of repetitive elements. 116 pp. Englisch. Seller Inventory # 9783846528129

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Daphne S. Cabianca

Published by LAP LAMBERT Academic Publishing (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP LAMBERT Academic Publishing, 2012. Paperback. Condition: Good. Ships with Tracking Number! INTERNATIONAL WORLDWIDE Shipping available. May not contain Access Codes or Supplements. May be ex-library. Shipping & Handling by region. Buy with confidence, excellent customer service!. Seller Inventory # 3846528129

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David Cooper, Meena Upadhhyaya

Published by Taylor & Francis

ISBN 10: 1859962440 ISBN 13: 9781859962442

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About this Item: Taylor & Francis. Hardcover. Condition: VERY GOOD. Very Good copy, cover and pages show some wear from reading and storage. Binding may have light creases. Lots of life left in these pages. Seller Inventory # 2963220201

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Daphne S. Cabianca

Published by LAP LAMBERT Academic Publishing (2012)

ISBN 10: 3846528129 ISBN 13: 9783846528129

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About this Item: LAP LAMBERT Academic Publishing, 2012. Paperback. Condition: New. Ships with Tracking Number! INTERNATIONAL WORLDWIDE Shipping available. Buy with confidence, excellent customer service!. Seller Inventory # 3846528129n

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Juliana Prügel

Published by AV Akademikerverlag (2013)

ISBN 10: 3639477774 ISBN 13: 9783639477771

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About this Item: AV Akademikerverlag, 2013. Paperback. Condition: Good. Ships with Tracking Number! INTERNATIONAL WORLDWIDE Shipping available. May not contain Access Codes or Supplements. May be ex-library. Shipping & Handling by region. Buy with confidence, excellent customer service!. Seller Inventory # 3639477774

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Cooper, David N.

Published by Garland/BIOS Scientific Publishers (2016)

ISBN 10: 1859962440 ISBN 13: 9781859962442

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About this Item: Garland/BIOS Scientific Publishers, 2016. Paperback. Condition: New. PRINT ON DEMAND Book; New; Publication Year 2016; Not Signed; Fast Shipping from the UK. No. book. Seller Inventory # ria9781859962442_lsuk

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David Cooper, Meena Upadhhyaya

Published by Taylor & Francis 2004-04-22 (2004)

ISBN 10: 1859962440 ISBN 13: 9781859962442

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About this Item: Taylor & Francis 2004-04-22, 2004. Condition: New. Brand new book, sourced directly from publisher. Dispatch time is 4-5 working days from our warehouse. Book will be sent in robust, secure packaging to ensure it reaches you securely. Seller Inventory # NU-TNFPD-00035310

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