T Cells in Arthritis (Progress in Inflammation Research)

P. Miossec; W.B. Van den Berg; G.S. Firestein

ISBN 10: 376435853X / ISBN 13: 9783764358532

Published by Birkhäuser, Germany, 1998

About this Item

376435853X. Pictorial board hardcover, from closed pharmaceutical company library. 246 pages, index, illustrated with charts, tables and diagrams. Outlines arguments and data suggesting that T cells may, or may not, be central players in the pathogenesis of chronic RA. While each author their own point of view, the reader can consider a balanced view of the critical questions. Text in English. Sticker mark, front board, contents clean, tight and bright.

Seller Inventory # 031541

Bibliographic Details

• Title: T Cells in Arthritis (Progress in Inflammation Research)
• Publisher: Birkhäuser, Germany
• Publication Date: 1998
• Language: English
• ISBN 10: 376435853X
• ISBN 13: 9783764358532
• Binding: Hardcover
• Condition: Very Good
• Book Type: Book
• Seller catalogs:
  • Medical Research

About this title

Synopsis

Rheumatoid arthritis (RA) is the most common and most severe form of inflammatory arthritis. The pathogenesis of RA has been the subject of intense research for several decades. The prevailing hypotheses have changed over the years, and have attempted to incorporate the most recent data. Although T cells represent an important component of the cells which infiltrate the joint synovium, their contribution at a late stage of the disease remains a matter of debate. The goal of this book is to outline the major arguments and data suggesting that T cells may, or may not, be central players in the pathogenesis of chronic RA. While each of the editors and authors has his/her own bias (as will be clear by reading the respective chapters), our hope is that the readers will enjoy a complete and balanced view of the critical questions and experiments. This is not just an intellectual exercise since the direction of future therapeutic interventions depends heavily on how one interprets the pathogenesis of RA and the contribution of T cells.