Beamish Robert (186 results)

Condition: Very Good. Most items will be dispatched the same or the next working day. A copy that has been read, but is in excellent condition. Pages are intact and not marred by notes or highlighting. The spine remains undamaged.

Hardcover. Condition: Good. 1996. It's a preowned item in good condition and includes all the pages. It may have some general signs of wear and tear, such as markings, highlighting, slight damage to the cover, minimal wear to the binding, etc., but they will not affect the overall reading experience.

Hardcover. Condition: Very Good.

New! Hard bound in laminated boards, illustrated w/graphs & tables, xiv + Pp353. Includes chapter references. As new. Proceedings of a Satellite Symposium of the Thirtieth International Physiological Congress July 8-11, 1986, in Winnipeg, Canada. 765 grams.

HrdCvr with dj, both are VG with minor staining on the dj, former owner's bookplate inside front cover, the proceedings of the Winnipeg Centennial Symposium held October 27-30, 1974 in Winnipeg, an intriguing book, 8vo, 192 p.

Hard Cover. Condition: Very Good. First Edition. a very good hardcover; an ex-library copy with usual markings; internally pages bright and clean; ---------- SATISFACTION GUARANTEED --------- FAST, COURTEOUS SERVICE ------ ALL ORDERS SHIPPED WITH DELIVERY CONFIRMATION; Size: 6 x 9". Ex-Library.

Hardcover. black-&-white illustrations (illustrator). 1st edition. pp. xlii, 588. Heavy 8vo. Boards very sl. bowed; else near fine condition. A title in the Progress in Experimental Cardiology series.

Condition: New.

Paperback. Condition: new. Paperback. Whenever the coronary flow is inadequate to provide enough oxygen to meet the energy demands of the tissue, the heart becomes ischemic. Manifestations of myocardial ischemia include depression in contractile activity, changes in metabolic pattern, abnormalities in ultrastructure, and alterations in membrane potential. Ischemic changes during the early phase are reversible but as the period of ischemia is extended, the injury becomes irreversible. The transition from reversible to irreversible ischemic injury is usually associated with some membrane defects. It is worthwhile to consider that the irreversible damage to the ischemic myocardium occurs when the sarcolemmal membrane is altered in suoh a way that it would promote 2 a net gain of ca + in the cardiac cell upon reinstitution of blood flow. Suoh a lesion could result when mechanisms for the entry as well as removal 2 of ca + from the myocardial oell become defective. In this regard, 2 depression of the sarcolemmal ca + pump would favour the oocurrenoe of 2 intracellular ca + overload. Furthermore, inhibition of the Na+-K+ pump would lead to elevation of myoplasmic Na+ which oould then increase the 2 2 intracellular concentration of ca + through the sarcolemmal Na+-ca + exchange mechanism. In faot recent studies have revealed an inhibition of 2 the sarcolemmal Na+-ca + exchange mechanism in the ischemic heart and this 2 change could also contribute towards the occurrence of intracellular ca + 2 overload. In this regard, 2 depression of the sarcolemmal ca + pump would favour the oocurrenoe of 2 intracellular ca + overload. In faot recent studies have revealed an inhibition of 2 the sarcolemmal Na+-ca + exchange mechanism in the ischemic heart and this 2 change could also contribute towards the occurrence of intracellular ca + 2 overload. Shipping may be from multiple locations in the US or from the UK, depending on stock availability.

Condition: New.

[1975]. (Hardcover) Fine in very good plus dust jacket. 192pp. Illustrated cloth. Illustrated title page, photographs, bibliography. The dust jacket has minor edgewear. Foreword by J.W. Burns. (Ideas, Aging, Communication, Economic Conditions, Education, Genetics, World Affairs).

Condition: As New. Unread book in perfect condition.

[0-88755-660-4] [1999]. (Trade paperback) Near fine. xi, 256pp. Maps, photographs, illustrations, appendices, notes, bibliography, index. Locale: Manitoba; Prairie Provinces; Western Canada. (Manitoba, Medicine--Manitoba).

Condition: New. In.

Paperback. Condition: New.

Hardcover. Condition: Very Good. No Jacket. Hardcover, No Jacket. Boards very clean, bumped at spine ends, bookplate inside, pages clean and binding firm, taken from the International Symposium on Heart Metabolism in Health and Disease.

Condition: New. pp. 372.

Brand new book. Fast ship. Please provide full street address as we are not able to ship to P O box address.

hardcover. Condition: Good. Connecting readers with great books since 1972! Used textbooks may not include companion materials such as access codes, etc. May have some wear or writing/highlighting. We ship orders daily and Customer Service is our top priority!

[0-88755-660-4] [1999]. (Trade paperback) Fine. xi, 256pp. Signed by the authors. Maps, photographs, illustrations, appendices, notes, bibliography, index. Locale: Manitoba; Prairie Provinces; Western Canada. (Manitoba, Medicine--Manitoba).

Paperback. Condition: Brand New. 367 pages. 9.30x6.20x0.90 inches. In Stock.

Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Whenever the coronary flow is inadequate to provide enough oxygen to meet the energy demands of the tissue, the heart becomes ischemic. Manifestations of myocardial ischemia include depression in contractile activity, changes in metabolic pattern, abnormalities in ultrastructure, and alterations in membrane potential. Ischemic changes during the early phase are reversible but as the period of ischemia is extended, the injury becomes irreversible. The transition from reversible to irreversible ischemic injury is usually associated with some membrane defects. It is worthwhile to consider that the irreversible damage to the ischemic myocardium occurs when the sarcolemmal membrane is altered in suoh a way that it would promote 2 a net gain of ca + in the cardiac cell upon reinstitution of blood flow. Suoh a lesion could result when mechanisms for the entry as well as removal 2 of ca + from the myocardial oell become defective. In this regard, 2 depression of the sarcolemmal ca + pump would favour the oocurrenoe of 2 intracellular ca + overload. Furthermore, inhibition of the Na+-K+ pump would lead to elevation of myoplasmic Na+ which oould then increase the 2 2 intracellular concentration of ca + through the sarcolemmal Na+-ca + exchange mechanism. In faot recent studies have revealed an inhibition of 2 the sarcolemmal Na+-ca + exchange mechanism in the ischemic heart and this 2 change could also contribute towards the occurrence of intracellular ca + 2 overload.

Hardcover. Condition: Good. Satisfaction 100% guaranteed.

Paperback. Condition: new. Paperback. Whenever the coronary flow is inadequate to provide enough oxygen to meet the energy demands of the tissue, the heart becomes ischemic. Manifestations of myocardial ischemia include depression in contractile activity, changes in metabolic pattern, abnormalities in ultrastructure, and alterations in membrane potential. Ischemic changes during the early phase are reversible but as the period of ischemia is extended, the injury becomes irreversible. The transition from reversible to irreversible ischemic injury is usually associated with some membrane defects. It is worthwhile to consider that the irreversible damage to the ischemic myocardium occurs when the sarcolemmal membrane is altered in suoh a way that it would promote 2 a net gain of ca + in the cardiac cell upon reinstitution of blood flow. Suoh a lesion could result when mechanisms for the entry as well as removal 2 of ca + from the myocardial oell become defective. In this regard, 2 depression of the sarcolemmal ca + pump would favour the oocurrenoe of 2 intracellular ca + overload. Furthermore, inhibition of the Na+-K+ pump would lead to elevation of myoplasmic Na+ which oould then increase the 2 2 intracellular concentration of ca + through the sarcolemmal Na+-ca + exchange mechanism. In faot recent studies have revealed an inhibition of 2 the sarcolemmal Na+-ca + exchange mechanism in the ischemic heart and this 2 change could also contribute towards the occurrence of intracellular ca + 2 overload. In this regard, 2 depression of the sarcolemmal ca + pump would favour the oocurrenoe of 2 intracellular ca + overload. In faot recent studies have revealed an inhibition of 2 the sarcolemmal Na+-ca + exchange mechanism in the ischemic heart and this 2 change could also contribute towards the occurrence of intracellular ca + 2 overload. Shipping may be from our Sydney, NSW warehouse or from our UK or US warehouse, depending on stock availability.

Hardcover. Condition: New. US SELLER SHIPS FAST FROM USA.

Condition: New.

Paperback. Condition: new. Paperback. Pathophysiology of Heart Failure brings together leading basic scientists and clinicians, presenting new approaches to this complex problem, involving cardiomyopathic processes and ischemia perfusion injury. The result is a synthesis of state-of-the-art information on molecular biology, cellular physiology and structure-function relationships in the cardiovascular system. The role which excess intracellular calcium plays in the genesis of cardiac dysfunction is described as a fundamental mechanism underlying heart failure; one which may lead to improved prevention and treatment. Audience: Clinical and experimental cardiologists will find the book a helpful source of ideas and inspiration. Pathophysiology of Heart Failure brings together leading basic scientists and clinicians, presenting new approaches to this complex problem, involving cardiomyopathic processes and ischemia perfusion injury. Shipping may be from multiple locations in the US or from the UK, depending on stock availability.

Condition: New.

Condition: New. In.

hardcover. Condition: New. In shrink wrap. Looks like an interesting title!