Florian Perner (73 results)
- Softcover
Seller: California Books, Miami, FL, U.S.A.California Books
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- Softcover
Seller: California Books, Miami, FL, U.S.A.California Books
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- Softcover
Seller: Books Puddle, New York, NY, U.S.A.Books Puddle
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- Softcover
Seller: California Books, Miami, FL, U.S.A.California Books
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- Softcover
Seller: California Books, Miami, FL, U.S.A.California Books
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- Softcover
Seller: California Books, Miami, FL, U.S.A.California Books
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- Softcover
Seller: Books Puddle, New York, NY, U.S.A.Books Puddle
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US$ 97.55
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- Softcover
Seller: preigu, Osnabrück, Germanypreigu
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US$ 47.42
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Taschenbuch. Condition: Neu. Régulation de la phosphatase du cycle cellulaire CDC25C dans la leucémie aiguë | Florian Perner | Taschenbuch | Französisch | 2025 | Editions Notre Savoir | EAN 9786208902780 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | A…nbieter: preigu.
- Softcover
Seller: Books Puddle, New York, NY, U.S.A.Books Puddle
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- Softcover
Seller: Books Puddle, New York, NY, U.S.A.Books Puddle
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Condition: New.
- Softcover
Seller: Books Puddle, New York, NY, U.S.A.Books Puddle
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- Softcover
Seller: preigu, Osnabrück, Germanypreigu
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Taschenbuch. Condition: Neu. Regolazione della fosfatasi del ciclo cellulare CDC25C nella leucemia acuta | Florian Perner | Taschenbuch | Italienisch | 2025 | Edizioni Sapienza | EAN 9786208902803 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter…: preigu.
Language: German
Published by Südwestdeutscher Verlag für Hochschulschriften, 2019
- Softcover
Seller: preigu, Osnabrück, Germanypreigu
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US$ 54.08
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Taschenbuch. Condition: Neu. Regulation der Zellzyklus-Phosphatase CDC25C in der akuten Leukämie | Florian Perner | Taschenbuch | 80 S. | Deutsch | 2019 | Südwestdeutscher Verlag für Hochschulschriften | EAN 9786202323307 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at…]preigu[dot]de | Anbieter: preigu.
- Hardcover
Seller: Books Puddle, New York, NY, U.S.A.Books Puddle
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US$ 151.95
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Condition: New.
- Hardcover
Seller: AHA-BUCH GmbH, Einbeck, GermanyAHA-BUCH GmbH
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US$ 106.01
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Buch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Das Praxisbuch für die tägliche Diagnostik am MikroskopNeben den klinischen und radiologischen Untersuchungen kommt der histopathologischen Untersuchung von Lungengewebeproben eine entscheidungsrelevante Bedeutung zu. In diagnostisch schwierigen oder zwei…felhaften Fällen kann durch die histologische Begutachtung betroffener Lungenareale eine Diagnosesicherung erzielt und therapeutisch relevante Differentialdiagnosen ausgeschlossen werden.Die Diagnostik nicht-neoplastischer Lungenerkrankungen ist oft eine große Herausforderung für den diagnostizierenden Pathologen. Unter den knappen Zeitressourcen aber auch angesichts der stetig zunehmenden Anzahl neu definierter bzw. weiter verfeinerter Entitäten, ist es kaum möglich, den Überblick zu behalten oder gar sämtliche wesentlichen Kriterien zur Diagnosestellung aus der Erinnerung abzurufen.Dieses Buch bietet eine kurze, übersichtliche Darstellung der einzelnen Krankheitsentitäten und der diagnostischen Kriterien für die Untersuchung von Lungengewebeproben.Es begleitet den Pathologen bei der täglichen Arbeit am Mikroskop und erläutert kurz und knapp, welche diagnostischen Kriterien im konkreten Fall greifen und welche Differentialdiagnosen zu beachten sind. Die Autoren legen Wert auf eine standardisierte und übersichtliche Darstellung der einzelnen Krankheitsentitäten. Fallstricken, die zunächst die Diagnose in die falsche Richtung lenken können, werden aufgezeigt.Algorithmen erlauben dem Leser, gezielt den korrekten diagnostischen Pfad einzuschlagen und zügig zur richtigen Diagnose zu finden.Ein einmaliges Praxishandbuch für die tägliche Diagnostik des Pathologen.
- Softcover
- Print on Demand
Seller: PBShop.store US, Wood Dale, IL, U.S.A.PBShop.store US
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US$ 54.95
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PAP. Condition: New. New Book. Shipped from UK. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
- Softcover
- Print on Demand
Seller: PBShop.store UK, Fairford, GLOS, United KingdomPBShop.store UK
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US$ 56.23
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PAP. Condition: New. New Book. Delivered from our UK warehouse in 4 to 14 business days. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
- Softcover
- Print on Demand
Seller: PBShop.store US, Wood Dale, IL, U.S.A.PBShop.store US
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US$ 54.98
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PAP. Condition: New. New Book. Shipped from UK. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
- Softcover
- Print on Demand
Seller: PBShop.store UK, Fairford, GLOS, United KingdomPBShop.store UK
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PAP. Condition: New. New Book. Delivered from our UK warehouse in 4 to 14 business days. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
Language: English
Published by Omniscriptum, Our Knowledge Publishing, 2025
- Softcover
- Print on Demand
Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermanyBuchWeltWeit Ludwig Meier e.K.
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Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Internal tandem duplications in the FLT3 receptor gene (FLT3-ITD) are found in approximately 30% of patients with acute myeloid leukemia (AML). This oncogene influences various functions of the AML cell and leads to uncontrolled pro…liferation. Clinically, FLT3-ITD is associated with a poor prognosis in patients with AML. In this study, the influence of FLT3-ITD on the phosphatase CDC25C, which as a cell cycle regulator significantly influences the control of cell division, was investigated. The experiments described here showed that CDC25C is phosphorylated by FLT3-ITD and activated in an uncontrolled manner. In the leukemia cell model, a primarily protective effect of CDC25C was shown in the presence of oncogenic stress. The genetically non-intact cells were increasingly prevented from dividing by CDC25C. However, the simultaneous presence of the FLT3-ITD mutation resulted in a loss of this protective function. The significance of the mechanism described here therefore lies in the control of the response of the leukemia cell to damage within the DNA. However, CDC25C does not have an oncogenic function in the classical sense. 64 pp. Englisch.
Language: Spanish
Published by Ediciones Nuestro Conocimiento Mai 2025, 2025
- Softcover
- Print on Demand
Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermanyBuchWeltWeit Ludwig Meier e.K.
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Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware 68 pp. Spanisch.
- Softcover
- Print on Demand
Seller: Majestic Books, Hounslow, United KingdomMajestic Books
Contact seller4-star sellerCondition: New
US$ 93.02
US$ 8.68 shippingShips from United Kingdom to U.S.A.Quantity: 4 available
Condition: New. Print on Demand.
- Softcover
- Print on Demand
Seller: Biblios, frankfurt am main, HESSE, GermanyBiblios
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US$ 99.20
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Condition: New. PRINT ON DEMAND.
- Softcover
- Print on Demand
Seller: CitiRetail, Stevenage, United KingdomCitiRetail
Contact seller5-star sellerCondition: New
US$ 61.88
US$ 49.41 shippingShips from United Kingdom to U.S.A.Quantity: 1 available
Paperback. Condition: new. Paperback. Internal tandem duplications in the FLT3 receptor gene (FLT3-ITD) are found in approximately 30% of patients with acute myeloid leukemia (AML). This oncogene influences various functions of the AML cell and leads to uncontrolled proliferation. Clinically, FLT3-ITD is associated with a poor p…rognosis in patients with AML. In this study, the influence of FLT3-ITD on the phosphatase CDC25C, which as a cell cycle regulator significantly influences the control of cell division, was investigated. The experiments described here showed that CDC25C is phosphorylated by FLT3-ITD and activated in an uncontrolled manner. In the leukemia cell model, a primarily protective effect of CDC25C was shown in the presence of oncogenic stress. The genetically non-intact cells were increasingly prevented from dividing by CDC25C. However, the simultaneous presence of the FLT3-ITD mutation resulted in a loss of this protective function. The significance of the mechanism described here therefore lies in the control of the response of the leukemia cell to damage within the DNA. However, CDC25C does not have an oncogenic function in the classical sense. This item is printed on demand. Shipping may be from our UK warehouse or from our Australian or US warehouses, depending on stock availability.
- Softcover
- Print on Demand
Seller: PBShop.store US, Wood Dale, IL, U.S.A.PBShop.store US
Contact seller5-star sellerCondition: New
US$ 54.98
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PAP. Condition: New. New Book. Shipped from UK. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
- Softcover
- Print on Demand
Seller: PBShop.store US, Wood Dale, IL, U.S.A.PBShop.store US
Contact seller5-star sellerCondition: New
US$ 54.98
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PAP. Condition: New. New Book. Shipped from UK. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
- Softcover
- Print on Demand
Seller: PBShop.store US, Wood Dale, IL, U.S.A.PBShop.store US
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PAP. Condition: New. New Book. Shipped from UK. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
- Softcover
- Print on Demand
Seller: buchversandmimpf2000, Emtmannsberg, BAYE, Germanybuchversandmimpf2000
Contact seller5-star sellerCondition: New
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Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Internal tandem duplications in the FLT3 receptor gene (FLT3-ITD) are found in approximately 30% of patients with acute myeloid leukemia (AML). This oncogene influences various functions of the AML cell and leads to uncontrolled prolife…ration. Clinically, FLT3-ITD is associated with a poor prognosis in patients with AML. In this study, the influence of FLT3-ITD on the phosphatase CDC25C, which as a cell cycle regulator significantly influences the control of cell division, was investigated. The experiments described here showed that CDC25C is phosphorylated by FLT3-ITD and activated in an uncontrolled manner. In the leukemia cell model, a primarily protective effect of CDC25C was shown in the presence of oncogenic stress. The genetically non-intact cells were increasingly prevented from dividing by CDC25C. However, the simultaneous presence of the FLT3-ITD mutation resulted in a loss of this protective function. The significance of the mechanism described here therefore lies in the control of the response of the leukemia cell to damage within the DNA. However, CDC25C does not have an oncogenic function in the classical sense.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 64 pp. Englisch.
- Softcover
- Print on Demand
Seller: PBShop.store UK, Fairford, GLOS, United KingdomPBShop.store UK
Contact seller5-star sellerCondition: New
US$ 56.23
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PAP. Condition: New. New Book. Delivered from our UK warehouse in 4 to 14 business days. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
- Softcover
- Print on Demand
Seller: PBShop.store UK, Fairford, GLOS, United KingdomPBShop.store UK
Contact seller5-star sellerCondition: New
US$ 56.23
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PAP. Condition: New. New Book. Delivered from our UK warehouse in 4 to 14 business days. THIS BOOK IS PRINTED ON DEMAND. Established seller since 2000.
