Joshi Pushpa (14 results)

Hardcover. Condition: Good. Dust Jacket Condition: Good. Some wear to covers, sticker on front free endpaper, otherwise text clean and solid; some wear and rubbing to dust jacket which is (as issued? ) partially glued to covers; 8vo 8" - 9" tall; 385 pages.

Hardcover. Condition: Good. THERE ARE NO TARIFFS OR CUSTOMS DUTIES ON BOOKS. A comprehensive collection of Gandhi's writings and speeches about women. This copy is one of 1,000 printed in 2002, following on from 2,000 printed in 1988, making it fairly scarce.The boards are bumped at the corners but the text is clean and tight. The dustjacket has some shelf wear to the edges and marks to the back cover. Overall a good copy.

Taschenbuch. Condition: Neu. Mitochondrial DNA abnormalities in patients with idiopathic myositis | Molecular, histological and biochemical analysis | Pushpa Raj Joshi | Taschenbuch | 132 S. | Englisch | 2015 | Südwestdeutscher Verlag für Hochschulschriften | EAN 9783838133768 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu.

Paperback. Condition: Brand New. 216 pages. 8.66x5.91x0.49 inches. In Stock.

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Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Mitochondrial DNA deletions are seen in patients with different forms of myositis, being most prominent in inclusion body myositis (IBM). However, the level of common deletion in myositis is very small. Real-time PCR analysis is the ideal method to quantify very less levels of deletion with higher sensitivity. Histologically, COX deficient muscle fibres and ragged-red muscle fibres are seen in patients with all three forms of myositis. Hence, these mitochondrial defects could be considered as precursors of pathogenesis in myositis. Multiple-cell real-time PCR analysis detected common deletion in both normal and COX negative fibres. Genarally, the degree of presence of common deletion in a fibre to become COX negative is roughly 80%, suggesting the argument that mitochondrial protein synthesis is impaired when a particular percentage of deletions in mtDNA is reached. However, the high levels of deletions in mtDNA alone do not seem to cause a severe COX deficiency and/or residual COX deficiency. Hence the defect in mitochondrial protein synthesis is predominantly by the reduction of wild-type mtDNA in affected cells. 132 pp. Englisch.

Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Joshi Pushpa RajStudied Microbiology at Tribhuwan University, Nepal and Master of Biomedical Engineering at University of Halle-Wittenberg, Germany. PhD leading to Dr. rer. medic. at Dept. of Neurology, University of Halle-Wittenberg.

Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Pushpa Dr.PUSHPA, Ph.D. - Geography Lecturer, Government Inter College Machhor, Almora Uttrakhand, India. Works on Urban-Geomorphology research. JYOTI JOSHI, P.hD., is an Assistant Professor, Department of Geography, Kumaun Universit.

Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Mitochondrial DNA deletions are seen in patients with different forms of myositis, being most prominent in inclusion body myositis (IBM). However, the level of common deletion in myositis is very small. Real-time PCR analysis is the ideal method to quantify very less levels of deletion with higher sensitivity. Histologically, COX deficient muscle fibres and ragged-red muscle fibres are seen in patients with all three forms of myositis. Hence, these mitochondrial defects could be considered as precursors of pathogenesis in myositis. Multiple-cell real-time PCR analysis detected common deletion in both normal and COX negative fibres. Genarally, the degree of presence of common deletion in a fibre to become COX negative is roughly 80%, suggesting the argument that mitochondrial protein synthesis is impaired when a particular percentage of deletions in mtDNA is reached. However, the high levels of deletions in mtDNA alone do not seem to cause a severe COX deficiency and/or residual COX deficiency. Hence the defect in mitochondrial protein synthesis is predominantly by the reduction of wild-type mtDNA in affected cells.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 132 pp. Englisch.

Taschenbuch. Condition: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Mitochondrial DNA deletions are seen in patients with different forms of myositis, being most prominent in inclusion body myositis (IBM). However, the level of common deletion in myositis is very small. Real-time PCR analysis is the ideal method to quantify very less levels of deletion with higher sensitivity. Histologically, COX deficient muscle fibres and ragged-red muscle fibres are seen in patients with all three forms of myositis. Hence, these mitochondrial defects could be considered as precursors of pathogenesis in myositis. Multiple-cell real-time PCR analysis detected common deletion in both normal and COX negative fibres. Genarally, the degree of presence of common deletion in a fibre to become COX negative is roughly 80%, suggesting the argument that mitochondrial protein synthesis is impaired when a particular percentage of deletions in mtDNA is reached. However, the high levels of deletions in mtDNA alone do not seem to cause a severe COX deficiency and/or residual COX deficiency. Hence the defect in mitochondrial protein synthesis is predominantly by the reduction of wild-type mtDNA in affected cells.

Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Pushpa Dr.PUSHPA, Ph.D. - Dozent fuer Geographie, Government Inter College Machhor, Almora Uttrakhand, Indien. Arbeitet in der Stadt-Geomorphologie-Forschung. JYOTI JOSHI, P.hD., ist Assistenzprofessorin an der Abteilung fuer Geographi.